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Tylenol poising



Cyanideis a monovalent compound containing a CN group. In the body, cyanidecauses death because it makes it incapable of utilizing oxygen tosustain life. Some of the common signs and symptoms associated withcyanide include confusion, general weakness, shortness of breath andvomiting and abdominal pain. Cases of cyanide poisoning requireimmediate medical attention. This essay will evaluate the mechanismof action of cyanide, it sources and also clarify whether oxygenationcan be used to manage oxygen poisoning.


Afterbinding to the cytochrome c oxidase, cyanide attaches to the ironwithin the protein complex hence inhibiting normal electron transportactivities. As a result, the iron in the protein cannot transportelectron to oxygen and hence halting ATP production (García, Gotor &ampRomero, 2014). Without ATP, electron transport cannot work, and thereis protein build up and deprive chemical energy, and this causesdeath

Victimsof cyanide poisoning are not able to carry out ATP energy productionefficiently. Once in the body, cyanide is easily broken down in theProtein A3 because it has a high affinity for it and this preventsoxygen from combining with hydrogen molecules. In the case study, thepatient died because oxygen contained in the blood is completelydifferent from the one used in ATP production.

Artificialrespiration of pure of oxygen would not entirely save cyanidevictims. It is only recommended because it increases the arterialsaturation of hemoglobin above normal levels (97.5%) and thisautomatically increases the amount of oxygen that dissolves in theplasma (Hampson, Piantadosi, Thom, &amp Weaver, 2012). Although themetabolic effects of oxygenation are small, it is recommended becauseit can prolong life and allow one to seek adequate treatment.Nitrates and thiosulfates should be used when conducting artificialrespiration because they increase the ability of blood to carryoxygen to the brain. Some of the common sources of cyanide poisoninginclude industries that use such chemicals in metal processing,electroplating and the manufacture of synthetic plastics. Some plantssuch as apricot and cassava also contain cyanide and bamboo shoots(Readeet al. 2012).


Itis evident that cyanide poisoning is lethal and immediate medicalattention is required. Cyanide causes death because it inhibitsnormal electron transport activities hence halting ATP formation.Although artificial respiration of pure oxygen is recommended, itonly prolongs the victim`s life. Sources of cyanide poisoning includeplants such as cassava and electroplating processes among others.


García,I., Gotor, C., &amp Romero, L. C. (2014). Beyond toxicity: aregulatory role for mitochondrial cyanide.&nbspPlantsignaling &amp behavior,&nbsp9(1),2015-27.

Hampson,N. B., Piantadosi, C. A., Thom, S. R., &amp Weaver, L. K. (2012).Practice recommendations for the diagnosis, management, andprevention of carbon monoxide poisoning. Americanjournal of respiratory and critical care medicine,&nbsp186(11),1095-1101.

Reade,M. C., Davies, S. R., Morley, P. T., Dennett, J., &amp Jacobs, I. C.(2012). Review article: management of cyanide poisoning.&nbspEmergencyMedicine Australasia,&nbsp24(3),225-238.